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Æåëåçîäåôèöèòíàÿ àíåìèÿ è äð. ãåìàòîëîãè÷åñêèå ïðîÿâëåíèÿ öåëèàêèè
Ïðèìåðíî â 5% ñëó÷àåâ ïðè÷èíîé ÆÄÀ ÿâëÿåòñÿ öåëèàêèÿ, îòñóòñòâèå ýôôåêòà îò ëå÷åíèÿ ïåðîðàëüíûìè ïðåïàðàòàìè è îòñóòñòâèå ïîäüåìà æåëåçà â êðîâè ïîñëå 2 ÷ èíòåðâàëà îò ìîìåíòà ïðèåìà 100 ìã æåëåçà ÿâëÿåòñÿ íàèáîëåå õàðàêòåðíûìè ïðèçíàêàìè ÆÄÀ íà ôîíå öåëèàêèè (â ñðàâíåíèè ñ ÆÄÀ íà ôîíå õåëèêîáàêòåðíîé èíôåêöèè - ðåôðàêòåðíîñòü ê ïåðîðàëüíûì ïð-òàì æåëåçà èìååòñÿ â 2/3 ñëó÷àåâ):
Our prospective study of IDA published in 2005 has now been extended from 150 to 325 patients. The prevalence of celiac disease remained unchanged at 5.2%. The 17 celiac patients in this series were indistinguishable from the rest of the anemic patients by their age, severity of iron deficiency anemia, or serum albumin. Serum cholesterol 151±36 was significantly lower in celiac disease than in the other IDA patients (180±41, p=0.007). Only one patient had diarrhea, one had stunted growth and 2 had constipation. The most consistent clinical feature was complete refractoriness to oral iron treatment. and the complete absence of an increment in serum iron two hours after oral iron loading with 100 mg ferrous sulphate. C. Hershko and J. Patz Ironing out the mechanism of anemia in celiac disease Haematologica, December 1, 2008; 93(12): 1761 - 1765. Äðóãèå ãåìàòîëîãè÷åñêèå ïðîÿâëåíèÿ öåëèàêèè: Äðóãèå äåôèöèòíûå àíåìèè, òðîìáîöèòîïåíèÿ è òðîìáîöèòîç, ëåéêî-íåéòðîïåíèÿ, êîàãóëîïàòèè, ãèïîñïëåíèçì, ïîâûøåííûé ðèñê ðàçâèòèÿ ëèìôîïðîëèôåðàòèâíûõ çàáîëåâàíèé - ïîäðîáíåå â ñâîáîäíîì äîñòóïå: [Ññûëêè äîñòóïíû òîëüêî çàðåãèñòðèðîâàííûì ïîëüçîâàòåëÿì ] [Ññûëêè äîñòóïíû òîëüêî çàðåãèñòðèðîâàííûì ïîëüçîâàòåëÿì ]
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Èñêðåííå, Âàäèì Âàëåðüåâè÷. |